Heparin promotes rapid fibrillation of the basic parathyroid hormone at physiological pH

August 17, 2022

Title

Heparin promotes rapid fibrillation of the basic parathyroid hormone at physiological pH

Author

Luca M. Lauth, Bruno Voigt, Twinkle Bhatia, Lisa Machner, Jochen Balbach, Maria Ott

Year

2022

Journal

FEBS Letters

Abstract

In acidic secretory granules of mammalian cells, peptide hormones including the parathyroid hormone are presumably stored in the form of functional amyloid fibrils. Mature PTH, however, is considerably positively charged in acidic environments, a condition known to impede unassisted self-aggregation into fibrils. Here, we studied the role of the polyanion heparin on promoting fibril formation of PTH. Employing ITC, CD spectroscopy, NMR, SAXS, and fluorescence-based assays, we could demonstrate that heparin binds PTH with submicromolar affinity and facilitates its conversion into fibrillar seeds, enabling rapid formation of amyloid fibrils under acidic conditions. In the absence of heparin, PTH remained in a soluble monomeric state. We suspect that heparin-like surfaces are required in vivo to convert PTH efficiently into fibrillar deposits.

Instrument

J-810

Keywords

peptide, hormones, heparin, amyloid fibrils