Molecular Mechanism on Forcible Ejection of ATPase Inhibitory Factor 1 from Mitochondrial ATP Synthase

April 20, 2023

Title

Molecular Mechanism on Forcible Ejection of ATPase Inhibitory Factor 1 from Mitochondrial ATP Synthase

Author

Kobayashi, Ryohei, Hiroshi Ueno, Kei-ichi Okazaki, and Hiroyuki Noji

Year

2023

Journal

Nature Communications

Abstract

IF1 is a natural inhibitor protein for mitochondrial FoF1 ATP synthase that blocks catalysis and rotation of the F1 by deeply inserting its N-terminal helices into F1. A unique feature of IF1 is condition-dependent inhibition although IF1 inhibits ATP hydrolysis by F1, IF1 inhibition is relieved under ATP synthesis conditions. To elucidate this condition-dependent inhibition mechanism, we have performed single-molecule manipulation experiments on IF1-inhibited bovine mitochondrial F1 (bMF1). The results show that IF1-inhibited F1 is efficiently activated only when F1 is rotated in the clockwise (ATP synthesis) direction, but not in the counterclockwise direction. The observed rotational-direction-dependent activation explains the condition-dependent mechanism of IF1 inhibition. Investigation of mutant IF1 with N-terminal truncations shows that the interaction with the γ subunit at the N-terminal regions is crucial for rotational-direction-dependent ejection, and the middle long helix is responsible for the inhibition of F1.

Instrument

V-660

Keywords

Enzyme mechanisms